Monday, 10 March 2014

Identification, Types, and Theorized-Behaviours for All Mental Stress

The designated subject commences beneath the very-professional wiggly-line.

OK, I promised I would make an addition today to the subject of Dopamine-and-Health, and I'm here delivering in the last hour (precisely last minute now!) of my time-zone. I feel bad thinking that someone might've checked at an earlier hour and been disappointed. My last several hours were used reducing an extreme pain-level of an atypical pain, and my several hours before that were used mostly foolishly telling myself that the pain wasn't worsening.

On the bright-painful side of things, I recognized and conceptualized a new factor/mechanism for that atypical pain, which might be useful for comprehension and treatment-aiding of certain cases that extend beyond just this pain.

Although smuggled in under the wire, I think that the implications and applications of the following Dopamine subject-addition is supreme, and I suppose it might even be the most significant-impacting item ever posted - an analysis of why that could be would form a quality and very subject-valuable future post, hopefully one that I deliver soon.

This wasn't what I'd planned to post tonight. I thought it might be positive to procedurally develop an understanding of related factors first, but I've got to meet my dumb self-imposed promised deadline, and this was the most to-go subject-piece. Due to the ad hoc circumstances, an iterative future-posting of this already seems likely to me.

The contrasting forms are accurate, their provided behaviours are theory, while the overall conclusion seems to be somewhat deadlocked by a list of extraneous considerations.

The Identity and Behaviour of Mental Stress:

Dopamine accommodates, and enables the formation of processes such as thoughts, interpretation of incoming data, and cellular-instruction, I'm theorizing, by bonding to a compound which provides core-data and is further assembled as a mental process.
If there is certain brain-region shortage of dopamine, there will be fewer dopamine molecules bonding to process compounds, and thought will be slower and not experience as much development (experiencing as less details included in a person's statements, or difficulty presenting a thought, and much more) Also, fewer processes will be engaged at any time, and those processes will have vulnerable and weakened constitutions. There can also be a backlog of processes when there is insufficient dopamine availability to commence their development and expansion, making responses delayed and staggered.
When there is an extreme shortage of prefrontal cortex and striatum dopamine, in addition to the aforementioned, the processing of senses is impaired and limited - Examples:

i) Five concurrent sounds might process as only one, making them lose their contexts, and their homogenized- unification lacking an identifiable meaning
ii)A body of text might be non-decipherable, appearing as just one item, and that item confusing and painful to look at; Other examples.
For here and now, I'm labeling these stress-experiences as imposing or encumbering stresses, to denote that they are of a very different nature to the next.

When no dopamine reserves are present in these same regions, mental processes will normally not commence until there is a minimum necessary amount of dopamine to bond to any of them.
However, in the event that there is no available specific-region dopamine for additional processes to be commenced and if a process is forcefully thrust into being processed ***, dopamine from already in-development processes will tear from its current compound to bond to the force-demanded process.
The compound from which the dopamine tore will be damaged, and constitute a more serious stress, and perhaps the typical conception. For here-now purpose, I'm labeling this type as Active Stress.

*** Likely causations include being startled / alarmed, aggressive arguments, if unexpectedly accused or confronted, when time-constricted with tasks, abuse, and many others.

Both these stress-types are likely to affect those of frequent anterior insula dopamine-dominance, but not those typically identifying as always or mostly prefrontal-etc dominant.

In a future post, I'll expound on why I suspect that mental Stress is 1:1 certain brain-region dopamine deficiency - making much mental ailment, including those age-related, likely to be the consequence of very long-term habitual deficiency, even if only a mild one.
When I expound on that conclusion, I'll provide a slew of reasons why this could be the singular-possible identity of virtually-all / maybe-all mental stress - having in-mind one possible similar-alternate instance that I've yet to spend time considering.

[  This doesn't cover preemption, or treatment considerations where a deficiency and mental stress is already existing  - treatment is vitally not simply supplementing a deficiency]


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