P.1 - Adrenaline abbreviation
P.2 - Neural pathways as pipelines
P.3 - Gauging Dopamine
P.4 - Insufficient Dopamine stresses the brain
P.5 - Subject Surmise
P.1
Dopamine and Adrenaline (hereby referring to both Epinephrine and Norepinephrine) are independently tasked, but their purposes are in tandem, and their consequential influence within a brain is relative to each other's status.
P.2
A brain's neural pathways can be considered as pipelines, synapses as gateways along the pipeline, and with there being adjustable openings between the gateways to process and transition information from one side of a synaptic gateway to the other.
The roles of Dopamine and Adrenaline within this concept are thus:
- Adrenaline / Noradrenaline determines the force with which information is flowing through the pipeline, and higher force equates to higher volume of information. The 'higher volume' is meant as relative to the amount of consideration a particular Anterior Insula normally produces.
- Adrenaline / Noradrenaline determines the force with which information is flowing through the pipeline, and higher force equates to higher volume of information. The 'higher volume' is meant as relative to the amount of consideration a particular Anterior Insula normally produces.
- Prefrontal Cortex Dopamine-availability determines how open or closed the gateways are, and so regulate how much information is being allowed to flow through the gateways, which are the synapses. It is always optimal that there is no resistance in information's flow through these, and any resistance is Mental Stress, and Dopamine-deficiency.
P.3
High Prefrontal Cortex Dopamine-availability with low Anterior Insula activity is as if having large pipelines with fully open gates - but little to flow through them.
Heavy Anterior Insula operation with low Prefrontal Cortex Dopamine results in less capacity for handling the flow of information through the brain, and when Adrenaline / Noradrenaline increases as Prefrontal Cortex Dopamine-availability languishes, control over thought becomes increasingly tenuous, and eventually will completely fail.
P.4
Weak control over information flowing through the Prefrontal Cortex is Mental Stress, and the beginnings for its ailments - such as a Psychosis.
Lower Prefrontal Cortex Dopamine means both lower control
over the systems regulating the flow of information in the brain, and over the meaning of the information, itself. It takes Dopamine-availability to make a Will-based determination upon what's presented in the Prefrontal Cortex from the Anterior Insula, and when there isn't sufficient Dopamine-availability...
When
information is flowing too fast and with insufficient Dopamine-availability for decisions to be made, considerations fall in default to an evaluation of False, and their False evaluations sees their considerations cycle back to the Anterior Insuala to be re-considered in their previous evolution, at the end of which they are sent again to the Prefrontal Cortex for a determination... and if this continues, the evolution of much considering will unravel like a string, in cyclic fashion downward with each progressive default-to-False evaluation.
P.5
As Adrenaline (Ep & Norep) increases, the considering of things in the Anterior Insula will occur at an accelerated pace. So, the more that Adrenaline increases, the more Prefrontal Cortex Dopamine-availability will be required to handle the greater volume of Anterior Insula consideration output.
Dopamine strengthens thought control, and is power of thought control. Adrenaline undermines that strength. Increasing Prefrontal Cortex Dopamine will relieve pressure within thought pipelines, which are the brain's neural pathways, while increasing Adrenaline will increase the pressure within those pathways.
Addendum: Reminder of Another Factor for Dopamine & Adrenaline Relations
Reflection: Reprisal of e=mc²... For Dopamine
Final Note: Non-temp Note on 'Dopamine and Adrenaline are Operational Antitheses'
Shrapnel
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